Overweight and obesity undoubtedly drive the prevalence of nonalcoholic fatty liver disease (NAFLD) in the population, promoting liver fat accumulation. There is also evidence that obesity may increase disease progression to nonalcoholic steatohepatitis (NASH), fibrosis, cirrhosis and eventually to hepatocellular carcinoma. Hence, weight loss is considered essential in overweight/obese patients with NAFLD to reduce the burden of the disease1; even a limited amount of weight loss is associated with decreased hepatic triglyceride content, measured by proton magnetic resonance spectroscopy, and remission of ultrasound-assessed steatosis, in a dose-dependent manner.