Inflammatory Bowel Disease (IBD) and its subtype Crohn’s disease (CD) arise due to a loss of tolerance to environmental antigens in genetically susceptible individuals. Longitudinal analysis of CD incidence has identified an inverse correlation between rates of communicable disease and autoimmune disorders, particularly in countries endemic for helminth infestation. A case report published by Broadhurst et al described the use of helminth eggs to treat an IBD patient with refractory disease. Induction of a type 2 immune response following helminth colonization promoted mucosal healing and achieved clinical remission.